Cerebral Intraparenchymal Hemorrhage
Dr. Robert M. Friedlander 4 Westchester Park Dr, White Plains, NY 10604 845•764•4353 Contact
Cost-Effectiveness of Postoperative Ketamine in Chiari Decompression
Dr. Robert M. Friedlander 4 Westchester Park Dr, White Plains, NY 10604 845•764•4353 Contact
Duraplasty Type as a Predictor of Meningitis and Shunting After Chiari I Decompression
Dr. Robert M. Friedlander 4 Westchester Park Dr, White Plains, NY 10604 845•764•4353 Contact
Predecompression and postdecompression cognitive andaffective changes in Chiari malformation type I
OBJECTIVE The role of the cerebellum in cognitive function and psychiatric symptoms is poorly understood and particularly understudied in patients with cerebellar pathologies such as Chiari malformation type I (CM-I).
Inhibition of mitochondrial protein import by mutant huntingtin
Chiari malformation, an anomaly of the posterior cranial
fossa, also known as Arnold–Chiari malformation, was described in autopsies
by Hans Chiari, an Austrian pathologist, in 1891. He defined four
distinct anatomical configurations in which the cerebellar tonsils (the most caudal
part of the cerebellum) protruded below the lower margin of the foramen magnum.1
Cerebral Intraparenchymal Hemorrhage: A Review, JAMA 2019
Importance Although spontaneous intraparenchymal hemorrhage (IPH) accounts for less than 20% of cases of stroke, it continues to be associated with the highest mortality of all forms of stroke and substantial morbidity rates.
Congenital and Acquired Chiari Syndrome
Chiari malformation, an anomaly of the posterior cranial
fossa, also known as Arnold–Chiari malformation, was described in autopsies
by Hans Chiari, an Austrian pathologist, in 1891. He defined four
distinct anatomical configurations in which the cerebellar tonsils (the most caudal
part of the cerebellum) protruded below the lower margin of the foramen magnum.1